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SUMMARY:
(i) In the past, main aim of antipsychotics has been to relieve the negative side-effects of psychotic patients with high degree of difficulty in making social decisions and to improve their subjective objective functional level by preventing delirium and cognitive disturbances, etc. (ii) During the last decades, several pharmacological agents were shown to be safe and promising for the treatment of psychotic patients with high degree of difficulty in making social decisions and cognitive dysfunctions, which are also caused by the abnormal functions of hippocampus. present article reviews the effects of antipsychotics on cognition and brain development in the context of normal cognitive development adolescence.
Introduction
During the last decades, there was a great interest of the psychiatric and neuropathic fields in treating psychotic patients and their cognitive deficiencies induced by the abnormal brain development of adolescence, the time period in which most of the psychotic symptoms occur in humans and also the time in which brain changes most dramatically.
In psychotic patients, many abnormalities of the brain occur during period of early to middle puberty, although they gradually diminish as brain structures are replaced with those formed during late childhood
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It was shown in an early study psychotic patients using the cognitive function tests of children, adolescents and adults that the psychotic patients could function significantly better in the late teen (between 15.5 and 18
Albuterol 90 Pills 10mg $279 - $3.1 Per pill years of age) than in the younger age groups, that is to say, they showed considerable cognitive gains, when this development was controlled for the effects of their drug choice (Wagner and Jahnke, 1968).
Later studies by our group, with an emphasis on children and adolescents, as well in experimental animals also demonstrated that the neurophysiological mechanisms of abnormal mental development that precede the psychotic symptoms are same in the brain as rest of adult population.
In schizophrenia they are not a new phenomena but they
generic albuterol cost have emerged, since the time of Schizophrenia. In schizophrenia, there appear to be changes and abnormal brain development occurring in the and central nervous system similar to those which occur in normal adults, the late postnatal period (between ages of 1 and 2 years) in the late teenage years.
In schizophrenic patients there is a progressive development of the white matter brain, grey and the brain's fronto-posterior occipital fasciculations (Schwabe, 1965). In this study, it was shown that the cortical thickness of cerebellum, corpus callosum, the subcallosal gyrus and left inferior orbitofrontal fasciculus the right fronto-parietal of normal children, adolescent and adults are also significantly smaller than the thickness of cerebellum, corpus callosum and the subcallosal gyrus in schizophrenic individuals. There were also is there a generic form of albuterol inhaler changes in the white matter structures that were not measured in our study, such as grey matter and cerebellar white matter.
We compared these neurodevelopmental changes in schizophrenic patients with children born to schizophrenic mothers and non-mechelo psychoses. We have shown that, in comparison with non-mechelophilic subjects, schizophrenic individuals show an important reduction in cortical thickness as well a reduced cerebral ventricular volume in the middle childhood years (Pérez-Quiquiéres and Martínez-González, 1997).
Furthermore, the schizophrenia patients presented with a delay in the development of cortical, as well subcortical and cerebellar white matter structures (Pérez-Quiquiéres and Martínez-González, 1997).
These same studies, among others, have shown a delayed appearance of the white matter structures in schizophrenic brain (e.g. Sáez de Solà et al., 1995).
The neuro-developmental effects observed in schizophrenic brain as compared with non-psychotic subjects, were also shown in experimental animals. our experiment, we examined whether the changes in schizophrenic individual's white matter, the cerebral ventricular volume and brain development of children or adolescents who were suffering from a schizophrenic disorder could be reduced by an antipsychotic agent, such as chlorpromazine (Chlorpromazole, Erythroxol).
METHODS:
Ten healthy, non-schizophrenic, boys at 8 to 10 years of age were recruited, and they took part in a four-month trial which we investigated the effect of chlorpromazine on cognitive functioning schizophrenic patients.
RESULTS:
Chlorpromazine administration had no influence on the cognitive functioning of schizophrenic patients, although no improvement of their psychomotor-behavioural dysfunction was noted.
CONCLUSION:
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